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Atherosclerosis
Volume 197, Issue 2
, Pages 564-571
, April 2008
Cyclophilin A differentially activates monocytes and endothelial cells: Role of purity, activity, and endotoxin contamination in commercial preparations
References
- . The cyclophilins. Genome Biol. 2005;6(7):226
- Distribution of the cyclosporine binding protein cyclophilin in human tissues. Immunology. 1991;72(3):399–404
- . Cyclophilin and peptidyl-prolyl cis-trans isomerase are probably identical proteins. Nature. 1989;337(6206):476–478
- Cyclophilin A binds to linear peptide motifs containing a consensus that is present in many human proteins. J Biol Chem. 2005;280(25):23668–23674
- . Calcineurin phosphatase activity in T lymphocytes is inhibited by FK 506 and cyclosporin A. Proc Natl Acad Sci USA. 1992;89(9):3686–3690
- . The chemistry of signal transduction.. Proc Natl Acad Sci USA. 1995;92(1):56–61
- . Identification of cyclophilin as a proinflammatory secretory product of lipopolysaccharide-activated macrophages. Proc Natl Acad Sci USA. 1992;89(8):3511–3515
- . Cyclophilin A as a novel biphasic mediator of endothelial activation and dysfunction. Am J Pathol. 2004;164(5):1567–1574
- . Cyclophilin A is secreted by a vesicular pathway in vascular smooth muscle cells. Circ Res. 2006;98(6):811–817
- Cyclophilin A is a proinflammatory cytokine that activates endothelial cells. Arterioscler Thromb Vasc Biol. 2004;24(7):1186–1191
- Dimethyl sulfoxide inhibits tissue factor expression, thrombus formation, and vascular smooth muscle cell activation: a potential treatment strategy for drug-eluting stents. Circulation. 2006;114(14):1512–1521
- Rapamycin, but not FK-506, increases endothelial tissue factor expression: implications for drug-eluting stent design. Circulation. 2005;112(13):2002–2011
- . Histamine induces tissue factor expression: implications for acute coronary syndromes. Circulation. 2005;112(3):341–349
- Celecoxib decreases endothelial tissue factor expression through inhibition of c-Jun terminal NH2 kinase phosphorylation. Circulation. 2005;111(13):1685–1689
- Different migration of vascular smooth muscle cells from human coronary artery bypass vessels. Role of Rho/ROCK pathway. J Vasc Res. 2007;44(2):149–156
- . Endotoxin contamination in recombinant human heat shock protein 70 (Hsp70) preparation is responsible for the induction of tumor necrosis factor alpha release by murine macrophages. J Biol Chem. 2003;278(1):174–179
- . Presence of cyclophilin A in synovial fluids of patients with rheumatoid arthritis. J Exp Med. 1997;185(5):975–980
- . Inflammation in atherosclerosis. Nature. 2002;420(6917):868–874
- TLR4-dependent NF-kappaB activation and mitogen- and stress-activated protein kinase 1-triggered phosphorylation events are central to Helicobacter pylori peptidyl prolyl cis-, trans-isomerase (HP0175)-mediated induction of IL-6 release from macrophages. J Immunol. 2006;177(11):7950–7958
- . Cyclosporine treatment reduces early atherosclerosis in the cholesterol-fed rabbit. Atherosclerosis. 1995;116(2):181–189
- . Chronic cyclosporine therapy impairs endothelium-dependent relaxation in the renal artery of the rat. J Am Soc Nephrol. 1992;2(8):1291–1297
- . Oxidative and nitrosative stress in kidney disease: a case for cyclosporine A. J Nephrol. 2005;18(4):453–457
- The risk of coronary artery disease after heart transplantation is increased in patients receiving low-dose cyclosporine, regardless of blood cyclosporine levels. Clin Cardiol. 1997;20(9):767–772
- . Cyclosporine and graft coronary artery disease after heart transplantation. Compr Ther. 2000;26(2):121–126
- Cyclosporine mitigates graft coronary artery disease in murine cardiac allografts: description and validation of a novel fully allogeneic model. J Heart Lung Transplant. 2005;24(4):446–453
PII: S0021-9150(07)00531-X
doi: 10.1016/j.atherosclerosis.2007.08.025
© 2007 Elsevier Ireland Ltd. All rights reserved.
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Atherosclerosis
Volume 197, Issue 2
, Pages 564-571
, April 2008
