Atherosclerosis
Volume 210, Issue 1 , Pages 48-50 , May 2010

The polymorphism Trp719Arg in the kinesin-like protein 6 is associated with the presence of late outgrowth endothelial progenitor cells in acute myocardial infarction

  • Siamak Davani

      Affiliations

    • Corresponding Author InformationCorresponding author. Tel.: +33 3 81 21 86 15; fax: +33 3 81 21 90 12.

Received 13 November 2009

References 

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  3. Stewart AF, Dandona S, Chen L, et al. Kinesin family member 6 variant Trp719Arg does not associate with angiographically defined coronary artery disease in the Ottawa Heart Genomics Study. J Am Coll Cardiol. 2009;53:1471–1472
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  15. Shiffman D, O’Meara ES, Bare LA, et al. Association of gene variants with incident myocardial infarction in the Cardiovascular Health Study. Arterioscler Thromb Vasc Biol. 2008;28:173–179
  16. Leone AM, Valgimigli M, Giannico MB, et al. From bone marrow to the arterial wall: the ongoing tale of endothelial progenitor cells. Eur Heart J. 2009;30:890–899
  17. Numaguchi Y, Sone T, Okumura K, et al. The impact of the capability of circulating progenitor cell to differentiate on myocardial salvage in patients with primary acute myocardial infarction. Circulation. 2006;114:I114–119
  18. Gnecchi M, Zhang Z, Ni A, Dzau VJ. Paracrine mechanisms in adult stem cell signaling and therapy. Circ Res. 2008;103:1204–1219

PII: S0021-9150(09)00998-8

doi: 10.1016/j.atherosclerosis.2009.11.041

Atherosclerosis
Volume 210, Issue 1 , Pages 48-50 , May 2010

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