- 1.1. Human coronary arteries without evidence of atherosclerosis and atherosclerotic coronary arteries are unable to synthesize cholesterol from acetate.
- 2.2. Synthesis of cholesterol esters is small in coronary arteries regardless of the presence or absence of atherosclerotic lesions.
- 3.3. Identical uptakes of cholesterol were found in normal and atherosclerotic coronary arteries.
- 4.4. Lipid synthesis in human saphenous veins perfused at a pressure of Hg does not differ from that in nonatherosclerotic coronary arteries or saphenous veins perfused at a pressure of .
- 5.5. Cholesterol uptake in saphenous veins perfused at a pressure of is lower than that in coronary arteries or saphenous veins perfused at arterial pressure (.
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☆Supported by grants from the Council for Tobacco Research, U.S.A., the Kenneth T. and Eileen L. Norris Foundation, the George and Miriam Gould Dock Fund, and the Robert E. and May R. Wright Foundation.