Objective: Autophagy has emerged as a cell survival mechanism critical for cellular homeostasis,
which may play a protective role in atherosclerosis. ATG16L1, a protein essential
for early stages of autophagy, has been implicated in the pathogenesis of inflammatory
diseases such as Crohn’s disease. However, it is unknown whether ATG16L1 is involved
in atherosclerosis. Our aim was to analyze ATG16L1 expression in carotid atherosclerotic
plaques in relation to markers of plaque vulnerability.
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Article Info
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EAS-0407
Identification
Copyright
© 2015 Published by Elsevier Inc.
