Objective: Autophagy has emerged as a cell survival mechanism critical for cellular homeostasis,
which may play a protective role in atherosclerosis. ATG16L1, a protein essential
for early stages of autophagy, has been implicated in the pathogenesis of inflammatory
diseases such as Crohn’s disease. However, it is unknown whether ATG16L1 is involved
in atherosclerosis. Our aim was to analyze ATG16L1 expression in carotid atherosclerotic
plaques in relation to markers of plaque vulnerability.
To read this article in full you will need to make a payment
Purchase one-time access:
Academic & Personal: 24 hour online accessCorporate R&D Professionals: 24 hour online accessOne-time access price info
- For academic or personal research use, select 'Academic and Personal'
- For corporate R&D use, select 'Corporate R&D Professionals'
Subscribe:
Subscribe to AtherosclerosisAlready a print subscriber? Claim online access
Already an online subscriber? Sign in
Register: Create an account
Institutional Access: Sign in to ScienceDirect
Article info
Publication history
EAS-0407
Identification
Copyright
© 2015 Published by Elsevier Inc.