Aim: Recent studies suggest high-density lipoproteins (HDL) and apolipoprotein A-I (apoA-I)
may lead to heart failure (HF) despite normal coronary arteries. We tested whether
low HDL cholesterol (HDLc), pro-inflammatory state and serum HDL cholesterol efflux
capacity (CEC) occurs during progressive decay of left ventricular (LV) function in
a swine model of dilated cardiomyopathy (DCM).
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EAS-0098
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Copyright
© 2015 Published by Elsevier Inc.
