Aim: Recent studies suggest high-density lipoproteins (HDL) and apolipoprotein A-I (apoA-I) may lead to heart failure (HF) despite normal coronary arteries. We tested whether low HDL cholesterol (HDLc), pro-inflammatory state and serum HDL cholesterol efflux capacity (CEC) occurs during progressive decay of left ventricular (LV) function in a swine model of dilated cardiomyopathy (DCM).
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