Objectives: Macrophage migration inhibitory factor (MIF) is a chemokine-like inflammatory mediator
that plays an important role in inflammatory diseases. MIF has been found to promote
atherogenesis in several models. However, recent data suggest that MIF has a complex
role in cardiovascular disease (CVD) including cardioprotective effects in models
of myocardial ischemia/reperfusion injury. Here, we studied the impact of Mif gene deletion in the ApoE-/- background .
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Article Info
Publication History
EAS16-1001 Novel Pathways in Atherosclerosis
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© 2016 Published by Elsevier Inc.
