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Correspondence| Volume 277, P227-228, October 2018

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Concerning “Comments and question on “Selective inhibition of endothelial NF-kB signaling attenuates chronic intermittent hypoxia-induced atherosclerosis in mice” ”

Published:June 16, 2018DOI:https://doi.org/10.1016/j.atherosclerosis.2018.06.862
Concerning “Comments and question on “Selective inhibition of endothelial NF-kB signaling attenuates chronic intermittent hypoxia-induced atherosclerosis in mice” ”
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      We have read with great interest the article by Song et al. [
      • Song D.
      • Fang G.
      • Mao S.Z.
      • Ye X.
      • Liu G.
      • Miller E.J.
      • Greenberg H.
      • Liu S.F.
      Selective inhibition of endothelial NF-kappaB signaling attenuates chronic intermittent hypoxia-induced atherosclerosis in mice.
      Atherosclerosis. 2018; 270: 68-75
      ] and their subsequent correspondence with Drs Della Schiava and Lermusiaux [
      • Della Schiava N.
      • Lermusiaux P.
      Comments and question on "Selective inhibition of endothelial NF-kappaB signaling attenuates chronic intermittent hypoxia-induced atherosclerosis in mice".
      Atherosclerosis. 2018; 272: 247
      ,
      • Song D.
      • Fang G.
      • Mao S.Z.
      • Ye X.
      • Liu G.
      • Miller E.J.
      • Greenberg H.
      • Liu S.F.
      Reply to: "Comments and question on "Selective inhibition of endothelial NF-kappaB signaling attenuates chronic intermittent hypoxia-induced atherosclerosis in mice"".
      Atherosclerosis. 2018; 272: 248
      ]. Based on the well-described intimate crosstalk between HIF-1 and NF-κB in several hypoxic conditions [
      • Taylor C.T.
      Interdependent roles for hypoxia inducible factor and nuclear factor-kappaB in hypoxic inflammation.
      J. Physiol. 2008; 586: 4055-4059
      ], the question by Drs. Della Schiava and Lermusiaux on whether such a crosstalk exists in chronic intermittent hypoxia (CIH)-induced atherosclerosis is very relevant.

      Keywords

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      References

        • Song D.
        • Fang G.
        • Mao S.Z.
        • Ye X.
        • Liu G.
        • Miller E.J.
        • Greenberg H.
        • Liu S.F.
        Selective inhibition of endothelial NF-kappaB signaling attenuates chronic intermittent hypoxia-induced atherosclerosis in mice.
        Atherosclerosis. 2018; 270: 68-75
        View in Article
        • Della Schiava N.
        • Lermusiaux P.
        Comments and question on "Selective inhibition of endothelial NF-kappaB signaling attenuates chronic intermittent hypoxia-induced atherosclerosis in mice".
        Atherosclerosis. 2018; 272: 247
        View in Article
        • Song D.
        • Fang G.
        • Mao S.Z.
        • Ye X.
        • Liu G.
        • Miller E.J.
        • Greenberg H.
        • Liu S.F.
        Reply to: "Comments and question on "Selective inhibition of endothelial NF-kappaB signaling attenuates chronic intermittent hypoxia-induced atherosclerosis in mice"".
        Atherosclerosis. 2018; 272: 248
        View in Article
        • Taylor C.T.
        Interdependent roles for hypoxia inducible factor and nuclear factor-kappaB in hypoxic inflammation.
        J. Physiol. 2008; 586: 4055-4059
        View in Article
        • Arnaud C.
        • Dematteis M.
        • Pepin J.L.
        • Baguet J.P.
        • Levy P.
        Obstructive sleep apnea, immuno-inflammation, and atherosclerosis.
        Semin. Immunopathol. 2009; 31: 113-125
        View in Article
        • Song D.
        • Fang G.
        • Greenberg H.
        • Liu S.F.
        Chronic intermittent hypoxia exposure-induced atherosclerosis: a brief review.
        Immunol. Res. 2015; 63: 121-130
        View in Article
        • Gras E.
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        • Pepin J.L.
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        • Godin-Ribuot D.
        Endothelin-1 mediates intermittent hypoxia-induced inflammatory vascular remodeling through HIF-1 activation.
        J. Appl. Physiol. 1985; 2016: 437-443
        View in Article

      Article info

      Publication history

      Published online: June 16, 2018
      Accepted: June 15, 2018
      Received: May 30, 2018

      Identification

      DOI: https://doi.org/10.1016/j.atherosclerosis.2018.06.862

      Copyright

      © 2018 Elsevier B.V. All rights reserved.

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