Background and Aims: Macrophages are one of the critical drivers of atherosclerotic plaque inflammation, which may be beneficial in early disease stages, but later more detrimental. The latter effects could potentially be halted by re-instructing plaque macrophages towards an anti-inflammatory, inflammation-resolving phenotype. Therefore, we aim to identify key genes in atherosclerosis that skew macrophages from pro- towards an anti-atherogenic phenotypes.
To read this article in full you will need to make a payment
Purchase one-time access:Academic & Personal: 24 hour online accessCorporate R&D Professionals: 24 hour online access
One-time access price info
- For academic or personal research use, select 'Academic and Personal'
- For corporate R&D use, select 'Corporate R&D Professionals'
Subscribe:Subscribe to Atherosclerosis
Already a print subscriber? Claim online access
Already an online subscriber? Sign in
Register: Create an account
Institutional Access: Sign in to ScienceDirect
Published online: August 03, 2019EAS19-0937
© 2019 Published by Elsevier Inc.