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Correction| Volume 328, P60-61, July 2021

Corrigendum to “Imbalanced insulin action in chronic overnutrition: Clinical harm, molecular mechanisms, and a way forward” [Atherosclerosis 247 (April 2016) 225–282]

  • Kevin Jon Williams
    Correspondence
    Corresponding author. Section of Endocrinology, Diabetes, and Metabolism, Department of Medicine, Temple University School of Medicine, Philadelphia, PA, USA.
    Affiliations
    Section of Endocrinology, Diabetes, and Metabolism, Department of Medicine, Temple University School of Medicine, Philadelphia, PA, USA

    Department of Molecular and Clinical Medicine, Sahlgrenska Academy of the University of Gothenburg, Gothenburg, Sweden
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  • Xiangdong Wu
    Affiliations
    Section of Endocrinology, Diabetes, and Metabolism, Department of Medicine, Temple University School of Medicine, Philadelphia, PA, USA
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Published:June 04, 2021DOI:https://doi.org/10.1016/j.atherosclerosis.2021.05.021
Corrigendum to “Imbalanced insulin action in chronic overnutrition: Clinical harm, molecular mechanisms, and a way forward” [Atherosclerosis 247 (April 2016) 225–282]
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      The Authors apologise for an error in the published Fig. 1 in the above article. The correct figure can be found below.
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      Published online: June 04, 2021

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      DOI: https://doi.org/10.1016/j.atherosclerosis.2021.05.021

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      © 2016 Elsevier B.V. All rights reserved.

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      • Imbalanced insulin action in chronic over nutrition: Clinical harm, molecular mechanisms, and a way forward
        AtherosclerosisVol. 247
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          The growing worldwide prevalence of overnutrition and underexertion threatens the gains that we have made against atherosclerotic cardiovascular disease and other maladies. Chronic overnutrition causes the atherometabolic syndrome, which is a cluster of seemingly unrelated health problems characterized by increased abdominal girth and body-mass index, high fasting and postprandial concentrations of cholesterol- and triglyceride-rich apoB-lipoproteins (C-TRLs), low plasma HDL levels, impaired regulation of plasma glucose concentrations, hypertension, and a significant risk of developing overt type 2 diabetes mellitus (T2DM).
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